Dr Rita Pedrosa

Centre for Tumour Biology
Postdoctoral Research Assistant
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Unpicking the molecular mechanisms underlying angiocrine signalling.

Upon DNA-damaging therapy, blood vessels produce a cocktail of cytokines and chemokines – the angiocrine signal. Our laboratory recently published evidence that FAK can control this angiocrine signal and that this can help sensitise tumours to DNA-damaging therapy.

My work will identify other players in the regulation of angiocrine signaling using a CRISPR screen. I will also explore the molecular mechanisms underlying how FAK controls angiocrine signaling.

See other researchers working on:

Angiogenesis
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